Humans have two sources of cholesterol: absorption from ingested food and de novo synthesis by the liver High levels of cholesterol in the blood correlates with cardiovascular disease, but cholesterol is required to regulate membrane fluidity and as a precursor for important molecules like steroids and bile acids. Thus, humans evolved several types of regulation of cholesterol levels. How does the human body compensate for excess consumption of cholesterol in the diet?

Eating a cholesterol-richmeal causes the hypothalamus, the hunger center of the brain, to signal satiety

Excess cholesterol and a lack of ATP inhibit the activity of HMG-CoA reductase in the liver to decrease cholesterol synthesis

Transcription of the HMG-CoA reductase gene decreases in the presence of high intracellular cholesterol

High cholesterol concentrations upregulate the number of serum transport proteins that deliver cholesterol to the liver for degradation.

Adipose cells switch to using cholesterol instead of glucose to produce ATP

The human body compensates for excess consumption of cholesterol in the diet when B. Excess cholesterol and a lack of ATP inhibit the activity of HMG-CoA reductase in the liver to decrease cholesterol synthesis.

Cholesterol refers to the fat that is in the body of an individual. Cholesterol can be found in the food that one gets from animals.

Cholesterol is important for the building of cells. It should be noted that

one doesn't compensate for cholesterol by eating a cholesterol-richmeal.

It should be noted that the human body compensates for excess consumption of cholesterol in the diet when there's excess cholesterol and a lack of ATP inhibit the activity of HMG-CoA reductase in the liver to decrease cholesterol synthesis.

It can also occur when proteolytic cleavage of HMG-CoA reductase leads to stimulation when intracellular cholesterol levels are high.

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